The pathophysiology for deficient 25(OH)D causing an atherogenic lipid profile is unclear. Inadequate dietary intake of vitamin D may reduce calcium absorption in the gastrointestinal system. Prior studies have shown that deficient dietary intake of calcium is associated with higher body weight and a more atherogenic lipid metabolism,53, 54 potentially due to increased lipid metabolism in the body or reduced intake of cholesterol in the gut. However, the observed association between 25(OH)D and dyslipidemia may be confounded by shared metabolic risk factors rather than a causal mechanism. Serum 25(OH)D has been shown to be decreased in obese patients, in whom sequestration of 25(OH)D in body fat reduces detectable 25(OH)D in the serum.55 This phenomenon results in an inverse association between BMI and 25(OH)D. Given that BMI is also associated with dyslipidemia, the presence of obesity may explain the observed association of 25(OH)D with dyslipidemia. Unfortunately, we did not have BMI available in our data set to adjust for this potentially confounding variable. There also appears to be an association between lipid-lowering medication usage and increases in 25(OH)D,56, 57 suggesting that the observed association between low 25(OH)D levels and dyslipidemia may be due to patients with lipid derangements not being on statin therapy.
Although vitamin D supplementation is inexpensive, widely available, and effective in raising serum 25(OH)D, clinical trials have yet to find any link between vitamin D supplementation and changes in lipid profile or reduced CVD mortality.15, 18, 58, 59, 60, 61 Mendelian randomization studies have used single nucleotide polymorphisms leading to variations in 25(OH)D to examine vitamin D's role in the development of an atherogenic lipid profile and CVD.49, 62, 63 These studies have shown evidence that single nucleotide polymorphisms causing a genetically increased RLP-C49 and BMI62 are associated with reduced 25(OH)D. This supports the notion that 25(OH)D may be a marker for overall health rather than an independent risk factor for CVD. As there is not currently any definitive evidence that vitamin D has a causal role in atherosclerotic CVD,21 the associations seen in observational studies may be due to confounding factors that impact both serum 25(OH)D and serum lipids
刚收到验血报告，我的坏胆固醇上了50岁后一直有些偏高（去年134，今年138）。但是，今年总胆固醇第一次过线（201）。家庭医生的护士打电话说需要吃药降坏胆固醇。我对吃药一直持谨慎态度（目前什么保健药都不吃），所以先来这里问问有经验的健友，有没有不吃药也能降低坏胆固醇的方法？如果有就先试试，不行再去开药～～～先谢谢啦！
报告上还有钠和维生素D过界，其它指标都挺正常：）
摘几个重要的指标：
SODIUM（钠）：145（正常：134－144）
GLUCOSE（血糖）：92（正常：65－99）
CALCIUM（钙）：9.4（正常：8.7-10.2）
TSH（甲状腺刺激激素）：3.69 （正常：0.45-4.5，这个前几年也高到6.73,差点要吃药）
TRIGLYCERIDES（甘油三酯）：93（正常：0－149）
CHOLESTEROL, TOTAL：201（正常：100－199）
HDL CHOLESTEROL（好胆固醇）：44 （正常：》39，这个也一直只是勉强达标）
LDL CHOLESTEROL （坏胆固醇）：138（正常：0－99，十来年中只有两年勉强达标）
VITAMIN D ：21.9（正常：30－100，既然不缺钙，需要再补VD吗？）
🔥 最新回帖
https://www.webmd.com/cholesterol-management/features/the-new-cholesterol-diet-oatmeal-oat-bran
每天1.5杯oats meal 即可降胆固醇
https://www.everydayhealth.com/infographics/how-much-oatmeal-you-need-lower-cholesterol/
🛋️ 沙发板凳
我的总胆固醇比你的高，我的HDL是60， 但Colestrol/HDL 比例是3.7，低于4.5，医生说不用吃药。
你的总胆固醇与好胆固醇的比例约高于4.5。。。
也超级喜欢做菜，帮做菜大师顶一下。我的血脂指标跟你差不多。一点小经验
一、吃橄榄油 二、吃牛油果。三吃粗粮 四少吃水果，锻炼是必须的。
听自制秘方大师的话再不扔蛋黄了，这是对的。
希望有经验的网友提供建议。在此谢谢那些花时间回复过我的网友，你们的回复我都仔细读了，很有帮助！
https://www.lipidjournal.com/article/S1933-2874(15)00384-0/fulltext
The pathophysiology for deficient 25(OH)D causing an atherogenic lipid profile is unclear. Inadequate dietary intake of vitamin D may reduce calcium absorption in the gastrointestinal system. Prior studies have shown that deficient dietary intake of calcium is associated with higher body weight and a more atherogenic lipid metabolism,53, 54 potentially due to increased lipid metabolism in the body or reduced intake of cholesterol in the gut. However, the observed association between 25(OH)D and dyslipidemia may be confounded by shared metabolic risk factors rather than a causal mechanism. Serum 25(OH)D has been shown to be decreased in obese patients, in whom sequestration of 25(OH)D in body fat reduces detectable 25(OH)D in the serum.55 This phenomenon results in an inverse association between BMI and 25(OH)D. Given that BMI is also associated with dyslipidemia, the presence of obesity may explain the observed association of 25(OH)D with dyslipidemia. Unfortunately, we did not have BMI available in our data set to adjust for this potentially confounding variable. There also appears to be an association between lipid-lowering medication usage and increases in 25(OH)D,56, 57 suggesting that the observed association between low 25(OH)D levels and dyslipidemia may be due to patients with lipid derangements not being on statin therapy.
Although vitamin D supplementation is inexpensive, widely available, and effective in raising serum 25(OH)D, clinical trials have yet to find any link between vitamin D supplementation and changes in lipid profile or reduced CVD mortality.15, 18, 58, 59, 60, 61 Mendelian randomization studies have used single nucleotide polymorphisms leading to variations in 25(OH)D to examine vitamin D's role in the development of an atherogenic lipid profile and CVD.49, 62, 63 These studies have shown evidence that single nucleotide polymorphisms causing a genetically increased RLP-C49 and BMI62 are associated with reduced 25(OH)D. This supports the notion that 25(OH)D may be a marker for overall health rather than an independent risk factor for CVD. As there is not currently any definitive evidence that vitamin D has a causal role in atherosclerotic CVD,21 the associations seen in observational studies may be due to confounding factors that impact both serum 25(OH)D and serum lipids
读过您的博客，受益匪浅。您以数据为准，让人信服。今后要多晒太阳。就是太爱美了，怕晒黑
那家的藏红花在减20刀哈
周围的朋友推荐的， 我老公以前胆固醇高， 坚持每天一个葡萄果， 半年后就降下来了， 没有吃药。希望对你有帮助！
不要在跑步机上跑步 走路 伤害膝盖
这种机器 牵动全身运动
我的经验：每天走路40 分钟，很有帮助。
这个称呼还是留给别人吧，我承受不起。感谢你阅读我的博客。华女怕黑害人不浅，健康远比美丽重要。
我的数据与你的接近，除了HDL 73， LDL 102，但 甘油三脂129
我建议你的饮食当中荤食的量再将低一点比例，做菜尽可能不要放糖，甜食我基本是不吃。唯一喜欢的是烤地瓜。
几个月前我的VD也是20，我当时就问医生是否胆固醇降太低所致？因为人体合成D需要胆固醇。她说没有直接关系，让我补D。不过她同意我减半降脂药的剂量。
是否吃药还要看你有没有其他危险因素，比如血压血糖，心脏，是否甲减等。没有危险因素的话可以暂时不吃药。你的生活方式已经很健康了，也不缺锻炼，所以如果LDL继续升高吃药是最有效的降脂途径。
我也是同样问题 需要降低血脂 胆固醇
服药十年了，跟你年龄相仿。
血压 每次去量医生都说非常好（70／110左右）；
血糖甲减，这次报告上仍然正常；
心率静处时65左右
一直以来就是这个维生素D和坏胆固醇出界，其它都很不错～～～自我感觉也是精力体力都是越来越好呢：）
还有，带了小米手环几个月，日平均深睡数据是两小时出头：）
谢谢！
你这个LDL/HDL也到了顶端了。下面是转贴：
由此可見，我們不能僅僅關注自己的總膽固醇（TC）有多低和高密度脂蛋白膽固醇（HDL-C）有多高，而且還應該關注總膽固醇與高密度脂蛋白比值是否正常，總膽固醇 / 高密度脂蛋白比值是目前用來衡量患者發生各種心腦血管疾病可能性常用的重要檢測指標。總的說來，當兩者比值<5時，提示患者並發心腦血管疾病的可能性比較小，當兩者比值>5時，提示患者罹患各種心腦血管疾病的可能性比較高。（總膽固醇與高密度脂蛋白的比值男性最好小于4.5，女性最好小于3.5）。
后来注意饮食，戒掉了我最喜欢的虾和鱿鱼，这两样胆固醇很高，生完孩子后降到300以下，我是肉食动物，必须吃肉，只是注意不吃油腻的肉就好，多吃含纤维的蔬菜，今年体检是220，我觉得很满意了
我没怎么运动也没怎么改良吃的，就早上想起来的时候喝些蜂蜜 bragg apple cider vinegar 的水几个月，偶尔早上来点 加柠檬汁的蜂蜜 bragg apple cider vinegar。 是坚持了好一阵子。
希望今年没啥变化，好怕见医生
是要补的，我们这 过50 才正常。20 实在太低了。
多吃点燕麦。黑木耳和维生素B也有帮助。
https://www.chicagotribune.com/lifestyles/health/chi-cholesterol-fda-warnings-20150214-story.html