Disease states associated with carotenoderma include hypothyroidism, diabetes mellitus, anorexia nervosa, nephrotic syndrome, and liver disease. In hypothyroidism and diabetes mellitus, the underlying mechanism of hypercarotenemia is thought to be both impaired conversion of beta-carotene into retinol and the associated increased serum lipids. Diabetes mellitus has also been associated with carotenoderma through disease-specific diets that are rich in vegetables.[8] In the nephrotic syndrome, the hypercarotenemia is related to the associated increased serum lipids, similar to the above entities.
It is of note that kidney dysfunction in general is associated with hypercarotenemia as a result of decreased excretion of carotenoids. Liver dysfunction, regardless of origin, causes hypercarotenemia as a result of the impaired conversion of carotenoids to retinol. This is of particular interest because jaundice and carotenoderma can coexist in the same patient. Anorexia nervosa causes carotenoderma mainly through diets that are rich in carotenoids and the associated hypothyroidism. It tends to be more common in the restricting subtype of this disease, and is associated with numerous other dermatologic manifestations, such as brittle hair and nails, lanugo-like body hair, and xerosis. Although Alzheimer's disease has been associated with carotenoderma in some reports, most studies on serum carotenoids in these patients show that their levels of carotenoids and retinol are depressed, and may be associated with the development of dementia.[9] A true association between Alzheimer's disease and carotenoderma is unclear at this time. There have been case reports in the literature of increased serum carotenoids and carotenoderma that is unresponsive to dietary measures, with a genetic defect in carotenoid metabolic enzymes proposed. Canthaxanthin and astaxanthin are naturally occurring carotenoids that are used in the British and US food industry to add color to foods such as sausage and fish. Canthaxanthin has been used in over-the-counter “tanning pills” in the United States and Europe, but is not currently Food and Drug Administration (FDA)-approved for this purpose in the United States because of its adverse effects. These include hepatitis, urticaria, aplastic anemia, and a retinopathy characterized by yellow deposits and subsequent visual field defects.[10]
Infants and small children are especially prone to carotenoderma because of the cooked, mashed, and pureed vegetables that they eat. Processing and homogenizing causes carotene to become more available for absorption. A small 2.5 ounce jar of baby food sweet potatoes or carrots contains about 400-500% of an infant's recommended daily value of carotene. In addition to that source of carotene, infants are usually prescribed a liquid vitamin supplement, such as Tri-Vi-Sol, which contains vitamin A.
来自同篇文章：“Carotenoderma can be divided into two major types, primary and secondary. Primary carotenoderma is that developing from increased oral ingestion of carotenoids, whereas secondary carotenoderma is caused from underlying disease states that increase serum carotenoids with normal oral intake of these compounds. "
由于脂溶性维生素在人体纯存期长，无法及时代谢，会造成中毒性反应
你不能利用的都要通过肝肾解毒，维生素A过量会造成肝中毒
这是慢性的，不是三两天看得出来
对你自己负责，不要听信民科不负责任的宣传
更不是只有胡萝卜才有维生素A
胡萝卜便宜，甜，甜的东西吃多了会有什么问题自己去想
给你一个链接 https://www.healthline.com/health/hypervitaminosis-a
你自己狗一下 vitamin A toxicity 看看是不是真的，非要有人中毒了才罢休吗？
但我要减少胡萝卜量。我还自己种了大量番茄，今年控制的不错，送人很多。希望身体里的维生素A没有超量。
谢谢你！
看来，我还应该再见一次医生，查维生素A。
反正我不信。
https://www.ncbi.nlm.nih.gov/pubmed/22431270
不可一概而论
https://en.wikipedia.org/wiki/Carotenosis
Disease states associated with carotenoderma include hypothyroidism, diabetes mellitus, anorexia nervosa, nephrotic syndrome, and liver disease. In hypothyroidism and diabetes mellitus, the underlying mechanism of hypercarotenemia is thought to be both impaired conversion of beta-carotene into retinol and the associated increased serum lipids. Diabetes mellitus has also been associated with carotenoderma through disease-specific diets that are rich in vegetables.[8] In the nephrotic syndrome, the hypercarotenemia is related to the associated increased serum lipids, similar to the above entities.
It is of note that kidney dysfunction in general is associated with hypercarotenemia as a result of decreased excretion of carotenoids. Liver dysfunction, regardless of origin, causes hypercarotenemia as a result of the impaired conversion of carotenoids to retinol. This is of particular interest because jaundice and carotenoderma can coexist in the same patient. Anorexia nervosa causes carotenoderma mainly through diets that are rich in carotenoids and the associated hypothyroidism. It tends to be more common in the restricting subtype of this disease, and is associated with numerous other dermatologic manifestations, such as brittle hair and nails, lanugo-like body hair, and xerosis. Although Alzheimer's disease has been associated with carotenoderma in some reports, most studies on serum carotenoids in these patients show that their levels of carotenoids and retinol are depressed, and may be associated with the development of dementia.[9] A true association between Alzheimer's disease and carotenoderma is unclear at this time. There have been case reports in the literature of increased serum carotenoids and carotenoderma that is unresponsive to dietary measures, with a genetic defect in carotenoid metabolic enzymes proposed. Canthaxanthin and astaxanthin are naturally occurring carotenoids that are used in the British and US food industry to add color to foods such as sausage and fish. Canthaxanthin has been used in over-the-counter “tanning pills” in the United States and Europe, but is not currently Food and Drug Administration (FDA)-approved for this purpose in the United States because of its adverse effects. These include hepatitis, urticaria, aplastic anemia, and a retinopathy characterized by yellow deposits and subsequent visual field defects.[10]
Infants and small children are especially prone to carotenoderma because of the cooked, mashed, and pureed vegetables that they eat. Processing and homogenizing causes carotene to become more available for absorption. A small 2.5 ounce jar of baby food sweet potatoes or carrots contains about 400-500% of an infant's recommended daily value of carotene. In addition to that source of carotene, infants are usually prescribed a liquid vitamin supplement, such as Tri-Vi-Sol, which contains vitamin A.
喜鹊的分享非常真诚，赞！
所以此类病人要少吃或者不吃
来自同篇文章：“Carotenoderma can be divided into two major types, primary and secondary. Primary carotenoderma is that developing from increased oral ingestion of carotenoids, whereas secondary carotenoderma is caused from underlying disease states that increase serum carotenoids with normal oral intake of these compounds. "
你引用的那段是归类在secondary carotenoderma里的。
我是觉得没必要大量吃胡萝卜的。饮食杂一点、多样化为好。
你这个人心态不对。
您怎么看我一点也不重要，每个人自己在干什么难道自己不清楚
https://bbs.wenxuecity.com/health/843240.html
jw2009 认为我自己种的这点简直就是毛毛雨。
生的一磅，炒熟后就一碗，一顿就干掉了（吓死我了！）但是，我这点和抗癌的比，真不算啥。人家是拿5 磅来榨汁（刚才吓的得到了一点舒缓）。
https://bbs.wenxuecity.com/health/843240.html
不管怎么说，我都不怪大家，大家都是好意。
看来癌症还真不好预防，走着走着，就走歪了。
服了！
虽然，，，但是，，，
因为吃太多胡萝卜，肚子太撑少吃别的营养物质，也是一种风险
如果你身体不适，还这么辛苦科普大家。
分享经历，别人吃不吃我管不着，没这个权利，没这个资格。我多次说过我不是医生，不是医疗界专业人员。我喜欢看网友的第我让那个网友去吃了？
一句话，你说的过头了，事实不是你说的那样。
我喜欢看网友的第一手资料，希望大家分享自己的经验，但我没叫别人去试啊。人家吃不吃我管不着，我公开多次说过我不是医生，根本没资格。怎么就变成我让别人去做实验了！
我以前确实不知道有人对胡萝卜过敏。有网友说：“ 我就对这个过敏。对生的差些，对熟的，很厉害 ”
我跟贴：”第一次听说。谢谢分享。”https://bbs.wenxuecity.com/health/864123.html
你提醒说维生素A多吃了有毒，没错，好事。我可没有叫别人吃很多胡萝卜啊。生胡萝卜维生素A，人体吸收不如熟的，不如加了油的，想必你知道。
大学的时候有个朋友，食堂吃饭从不买土豆和白菜（这两菜我都爱吃）。有一天终于忍不住问他，答曰：这辈子再也不想吃它们，从小到大，冬天时只有这两菜，吃到吐。