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脑干肿瘤患者来美求医
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最新回复:2024年10月19日 10点15分 PT
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楼主 (北美华人网)
我的朋友患有脑干弥漫性中线胶质瘤。他要先在国内接受为期6周的放疗,然后再来美国做进一步的治疗。在这里我恳请大家施与援手,帮助他寻找在治疗他所患疾病方面经验丰富业内排名靠前的知名医生。
非常感谢大家的关注!
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病理报:
特殊染色:免疫组化结果: GFAP (+), 01ig-2 (+), IDH1 R132H (-), ATRX (+), p53 (+), Ki-67 (约10-20%), CD34 (血管+), H3K27M (+), H3K27me3(表达缺失), EZHIP (-), NeuN (神经元+).
病理诊断: (脑干)活体检本全部取材。弥漫性胶质瘤,细胞密度中等,可见核异形,偶见核分裂像,结合部分及免疫组化结果 (H3K27me3核表达缺失,H3K27M 阳性), 符合弥漫中线胶质瘤, H3 K27变异型, CNS WHO 4级。
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Pathology Report
Special dyeing: Immunohistochemical results: GFAP (+), 0lig-2 (+), IDH1 R132H(-), ATRX(+), p53 (+), Ki -67 (about 10-20%), CD34 (vascular +), H3K27M(+), H3K27me3 (expression deficiency), EZHIP (-), NeuN (neuron +).
Pathological diagnosis: (brain stem) biopsy is all based on materials. Diffuse glioma, medium cell density, visible nuclear heterotype, occasionally seen nuclear division image, binding site and immunohistochemical results (H3K27me3 nuclear expression missing, H3K27M positive), consistent with diffuse midline glioma, H3 K27 variant heterotype, CNS WHO Level 4.
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非常感谢大家的关注!
--------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------
病理报:
特殊染色:免疫组化结果: GFAP (+), 01ig-2 (+), IDH1 R132H (-), ATRX (+), p53 (+), Ki-67 (约10-20%), CD34 (血管+), H3K27M (+), H3K27me3(表达缺失), EZHIP (-), NeuN (神经元+).
病理诊断: (脑干)活体检本全部取材。弥漫性胶质瘤,细胞密度中等,可见核异形,偶见核分裂像,结合部分及免疫组化结果 (H3K27me3核表达缺失,H3K27M 阳性), 符合弥漫中线胶质瘤, H3 K27变异型, CNS WHO 4级。
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Pathology Report
Special dyeing: Immunohistochemical results: GFAP (+), 0lig-2 (+), IDH1 R132H(-), ATRX(+), p53 (+), Ki -67 (about 10-20%), CD34 (vascular +), H3K27M(+), H3K27me3 (expression deficiency), EZHIP (-), NeuN (neuron +).
Pathological diagnosis: (brain stem) biopsy is all based on materials. Diffuse glioma, medium cell density, visible nuclear heterotype, occasionally seen nuclear division image, binding site and immunohistochemical results (H3K27me3 nuclear expression missing, H3K27M positive), consistent with diffuse midline glioma, H3 K27 variant heterotype, CNS WHO Level 4.