I've been thinking and telling others. I live in NY and work in Nassau county. Beginning of January EVERYONE and when I say EVERYONEi mean ok 80% of people I work with, friends, family including my son, his classmates and the parents. We ALL had fever and flu-like symptoms. I swear my gut keeps telling me we have had this covid circulating when China was announcing they had a problem on the rise. Too many ppl are living in the dark and too many ppl are believing and following everything the government is saying. Time for ppl to wake up!!!
The same here! We were at Seattle, though. All the kids and teachers were experiencing flu-like symptoms, but it was there for weeks. My kid and I both coughed so hard but no runny nose... I talked with a senior teacher who was in her 60s. She had never had a flu like this bad... She felt fatigued all the time. School had even sent out warnings to families not bring the kids over if having had any fever in 24 hours
Nature Medicine 3/17/2020 发表了“The proximal origin of SARS-CoV-2”. 作者 Kristian G. Andersen, Andrew Rambaut, W. Ian Lipkin, Edward C. Holmes & Robert F. Garry 。 这个杂志权威性如何？ https://www.nature.com/articles/s41591-020-0820-9 Conclusions In the midst of the global COVID-19 public-health emergency, it is reasonable to wonder why the origins of the pandemic matter. Detailed understanding of how an animal virus jumped species boundaries to infect humans so productively will help in the prevention of future zoonotic events. For example, if SARS-CoV-2 pre-adapted in another animal species, then there is the risk of future re-emergence events. In contrast, if the adaptive process occurred in humans, then even if repeated zoonotic transfers occur, they are unlikely to take off without the same series of mutations. In addition, identifying the closest viral relatives of SARS-CoV-2 circulating in animals will greatly assist studies of viral function. Indeed, the availability of the RaTG13 bat sequence helped reveal key RBD mutations and the polybasic cleavage site.
The genomic features described here may explain in part the infectiousness and transmissibility of SARS-CoV-2 in humans. Although the evidence shows that SARS-CoV-2 is not a purposefully manipulated virus, it is currently impossible to prove or disprove the other theories of its origin described here. However, since we observed all notable SARS-CoV-2 features, including the optimized RBD and polybasic cleavage site, in related coronaviruses in nature, we do not believe that any type of laboratory-based scenario is plausible.
More scientific data could swing the balance of evidence to favor one hypothesis over another. Obtaining related viral sequences from animal sources would be the most definitive way of revealing viral origins. For example, a future observation of an intermediate or fully formed polybasic cleavage site in a SARS-CoV-2-like virus from animals would lend even further support to the natural-selection hypotheses. It would also be helpful to obtain more genetic and functional data about SARS-CoV-2, including animal studies. The identification of a potential intermediate host of SARS-CoV-2, as well as sequencing of the virus from very early cases, would similarly be highly informative. Irrespective of the exact mechanisms by which SARS-CoV-2 originated via natural selection, the ongoing surveillance of pneumonia in humans and other animals is clearly of utmost importance.
但也有另一种可能是中共实际已发动生化战争 习一直用这种总有刁民要害朕的方法拖延时间把它粉饰成权斗呢？
当了半天一尊，外交部的发言人也不是他的人？这。。。。
how convenient
习近平不控制外交以及海外部署，which is 江派 或者更多的是王岐山控制
这玩意跟美军投毒一样没有可信度
我的手下是个美国女孩子，家庭是铁杆共和党和川普的拥护者， 今天她和我说， 她怀疑病毒在美国去年十月十一月就有了， 她的几个亲人当时生病，症状和目前的新冠非常类似， 去看医生， 测了流感， 都是NEGATIVE, 病了好几个星期才好， 现在她家里人都怀疑他们当时得的就是新冠。
所以也不是只有国内的人才有这种猜测。
神经病啊，
如果真的是新冠，以新冠的传播速度，一个月就可以把医护人员放倒一片，
以欧洲和美国的亲密程度，意大利应该去年十月十一月就变成现在这样了
拿个电子烟致死传染给了同住不用电子烟的人的例子来看看？
这个造谣要保存！
你有证据说我造谣吗， 你有理性讨论的能力吗？
哈哈第一句 说的够直白
sars基本控制在国内了啊，2.0全球蔓延，还有who帮助隐瞒，说实在，就算病毒不是人造/泄漏，回头各国缓过来了中国都有大麻烦
Possible，从武汉缺口罩的处理来看都比较合理
我本来以为只是病毒泄露。你一说投毒，那就基本知道是谁干的了吧。和某铊投毒如出一辙。
祝wm外宣出门被车撞死
不是已經有2000多萬人感染流感了？死亡2萬多。。。。。這裡面有多少是新冠肺炎你確定沒有？沒人去開棺驗屍嚴明真相的。。。。這麼明顯的疑點，就是沒人去查！！！為什麼？
可不可以把片子传上来看一下呢？
先提供了再说。并且他家人有没有相同症状呢？
如果属实的话，这局也是自己人做的，我党伟光正形象彻底完蛋，还想继续干下去吗？
我的国的宣传还是骗了一批没头脑的，当然大外宣的可能性也很大。这件事太大，个体最好不要甩锅，扛不住的
网上用这种捕风捉影的也不知哪来的帖子，带节奏，转风向
王岐山是习插队就认识的，也是一手被习提拔上来，说他俩不是穿一条裤子，鬼都不信。
自己去微博看哈
同学，习开会，王都没参加。 任志强是王的关系比较好的，也被抓了
悲哀
你手下的话你也信
基本是这样
LOL. 这个帖子是考验智商是不是大于50的帖子。
那个流感与电子烟肺炎的确很迷惑，但同时更奇怪的是如果是同一个病症，为什么没有大规模传染成人或者老人，没有大规模死人。
如果硬要牵强解释 ，可能的理论就是这个病毒在美国时的菌株是比S型更低毒的，到了中国变异成烈性的,就象S型根本上到不了意大利,只有L型着陆.。
我感觉解释需要更多的FACTS.
Conclusions
In the midst of the global COVID-19 public-health emergency, it is reasonable to wonder why the origins of the pandemic matter. Detailed understanding of how an animal virus jumped species boundaries to infect humans so productively will help in the prevention of future zoonotic events. For example, if SARS-CoV-2 pre-adapted in another animal species, then there is the risk of future re-emergence events. In contrast, if the adaptive process occurred in humans, then even if repeated zoonotic transfers occur, they are unlikely to take off without the same series of mutations. In addition, identifying the closest viral relatives of SARS-CoV-2 circulating in animals will greatly assist studies of viral function. Indeed, the availability of the RaTG13 bat sequence helped reveal key RBD mutations and the polybasic cleavage site.
The genomic features described here may explain in part the infectiousness and transmissibility of SARS-CoV-2 in humans. Although the evidence shows that SARS-CoV-2 is not a purposefully manipulated virus, it is currently impossible to prove or disprove the other theories of its origin described here. However, since we observed all notable SARS-CoV-2 features, including the optimized RBD and polybasic cleavage site, in related coronaviruses in nature, we do not believe that any type of laboratory-based scenario is plausible.
More scientific data could swing the balance of evidence to favor one hypothesis over another. Obtaining related viral sequences from animal sources would be the most definitive way of revealing viral origins. For example, a future observation of an intermediate or fully formed polybasic cleavage site in a SARS-CoV-2-like virus from animals would lend even further support to the natural-selection hypotheses. It would also be helpful to obtain more genetic and functional data about SARS-CoV-2, including animal studies. The identification of a potential intermediate host of SARS-CoV-2, as well as sequencing of the virus from very early cases, would similarly be highly informative. Irrespective of the exact mechanisms by which SARS-CoV-2 originated via natural selection, the ongoing surveillance of pneumonia in humans and other animals is clearly of utmost importance.